Amish, Mennonite, and Hutterite
Genetic Disorder Database

Corticosterone Methyloxidase Type 1 Deficiency

Disorder
OMIM #: #203400  (Click to access OMIM database)
Disorder: Corticosterone Methyloxidase Type 1 Deficiency 
Also known as: CMO I DEFICIENCY
ALDOSTERONE DEFICIENCY I
HYPERRENINEMIC HYPOALDOSTERONISM FAMILIAL, 1
ALDOSTERONE DEFICIENCY DUE TO DEFECT IN STEROID 18-HYDROXYLASE
18-HYDROXYLASE DEFICIENCY
STEROID 18-HYDROXYLASE DEFICIENCY 
Clinical
Phenotype: dehydration, failure to thrive, vomiting, intermittent fever, hyponatremia, hyperkalemia, feeding difficulties, hypotension, decreased serum aldosterone, increased serum corticosterone, increased serum renin, decreased serum 18-hyroxycorticosterone 
Seen In: Amish
Old Order Mennonite
Old Colony Mennonite
Unknown/Other Mennonite
Hutterite
Remarks: Autosomal recessive  
Mutations
1   Amish  
Gene: CYP11B2
Base Change: c.104_109delinsG
Amino Acid Change: p.Val35Glyfs*3
Last updated: 2022-11-03 

References
Mitsuuchi Y, Kawamoto T, Miyahara K, Ulick S, Morton DH, Naiki Y, Kuribayashi I, Toda K, Hara T, Orii T, Yasuda K, Miura K, Yamamoto Y, Imura H, and Shizuta Y. (1993) Congenitally defective aldosterone biosynthesis in humans: inactivation of the P-450C18 gene (CYP11B2) due to nucleotide deletion in CMO I deficient patients. Biochem Biophys Res Commun 190(3): 864-869.
PubMed ID: 8439335 

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